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Hypoxia-Induced BACH1 Elevation Drives Pulmonary Hypertension via Activation of TGFBR2/SMAD Signaling

By MedXY|Jul 15, 2026

BACH1 stabilization under hypoxia promotes pulmonary hypertension by enhancing TGFBR2/SMAD pathway activity in pulmonary artery smooth muscle cells, suggesting BACH1 as a promising therapeutic target for PH.

Impact of Short-Term High-Altitude Exposure on Right Ventricular Load and Oxygen Delivery in Pulmonary Vascular Disease Patients

Cardiologyhigh altitude

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Hypoxia-Induced BACH1 Elevation Drives Pulmonary Hypertension via Activation of TGFBR2/SMAD Signaling

Impact of Short-Term High-Altitude Exposure on Right Ventricular Load and Oxygen Delivery in Pulmonary Vascular Disease Patients

Unveiling the Phenotype of Unclassified Pulmonary Hypertension: A Hidden Spectrum of Early Heart Failure with Preserved Ejection Fraction

CT Small-Artery Loss Tracks Severe Pulmonary Hypertension in COPD and Fibrosing ILD More Closely Than Parenchymal Damage

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The BMPR2 Nexus: How Breast Cancer Acts as a Second Hit for Pulmonary Hypertension