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Long-term cumulative exposure to wildfire smoke PM2.5 significantly increases cardiovascular hospitalizations among U.S. older adults, with notable risks in ischemic heart disease and arrhythmias, highlighting the need for targeted public h
Chronic exposure to fine particulate matter (PM2.5) is linked to epicardial coronary endothelial dysfunction, an early marker of coronary artery disease, as demonstrated by invasive coronary vasoreactivity testing.
High exposure to ambient NO2 and O3 air pollutants is associated with approximately one year earlier natural menopause, highlighting environmental risks to women’s reproductive aging.
Long-term exposure to particulate matter and lack of green space were associated with increased risk of prediabetes, especially impaired glucose tolerance phenotypes, and reduced remission of isolated impaired fasting glucose.
This review synthesizes evidence linking long-term exposure to PM2.5 constituents—particularly elemental carbon, organic carbon, and nitrate—with increased coronary heart disease and acute myocardial infarction risks in China, emphasizing t
This large-scale study links specific PM2.5 components, especially chloride, black carbon, and sulfate, to increased long-term cardiovascular disease risk, emphasizing targeted pollution control for better heart health.
A large U.S. study found that even PM2.5 levels below current federal standards were linked to higher hypertension hospitalization risk in older adults, especially among women and some regional and socioeconomic groups.
Among more than 26 million older U.S. adults, even PM2.5 levels below current standards were linked to higher hypertension hospitalization risk, suggesting no clearly safe low-exposure threshold.
Portable HEPA air cleaners plus motivational interviewing improved symptoms and quality of life in smokers with COPD by reducing indoor particulate matter, though lung function and exacerbations did not significantly change.
A study of 1,454 older adults with metabolic syndrome reveals that long-term exposure to air pollutants like black carbon, PM2.5, and NO2 is associated with increased fat mass and decreased lean mass, persisting over three years, particular
Recent evidence from a large-scale Swedish study reveals that long-term exposure to fine and coarse particulate matter and nitrogen dioxide significantly increases the risk of Motor Neuron Disease and accelerates functional decline and mort
A retrospective analysis of the ECHO cohort reveals that prenatal exposure to fine particulate matter (PM2.5) is associated with lower birth weight for gestational age, with critical windows of susceptibility identified as early as the firs
A prospective study reveals that PM2.5 and ozone exposure significantly exacerbate cardiac dysfunction and adverse remodeling in breast cancer patients receiving anthracyclines or trastuzumab, doubling the risk of clinical LVEF decline.
A comprehensive Swedish study reveals that long-term exposure to particulate matter and nitrogen dioxide significantly increases the risk of Motor Neuron Disease and accelerates functional decline, even in regions with relatively low pollut
This study explores how changes in residential environments affect childhood BMI, focusing on air pollution, built environment, and socioeconomic factors across cohorts in the Netherlands, Sweden, and Czech Republic.
This study links childhood exposure to outdoor air pollution with increased allostatic load, reflecting multi-system physiological stress, whereas road traffic noise showed no significant effect.
This review synthesizes recent evidence linking long-term air and noise pollution exposure with increased incidence of valvular heart disease, highlighting findings from a large UK Biobank cohort study, underlying mechanisms, and clinical i
This review synthesizes evidence linking long-term air pollution and road traffic noise exposure with antidepressant medication dispensing, highlighting sex-specific associations and implications for mental health prevention strategies.
A population-based study reveals that midlife exposure to air pollutants such as nitrogen dioxide and particulate matter is associated with cognitive decline and brain structural alterations by age 69-71, highlighting long-term neurological
This population-based study reveals that midlife exposure to nitrogen dioxide and particulate matter correlates with poorer cognitive function and adverse brain structural changes in later life, underscoring the neurocognitive risks of air